Parkinson's disease, neurodegeneration, neuroprotection, apoptotsis, autophagy, mitochondria, lysosome, inclusions, alpha-synuclein
1. Recasens A., Dehay B., Bové J., Carballoand#8208;Carbajal I., Dovero S., Pérez A., Fernagut P.O., Blesa J., Parent A., Perier C., Farińas I, Obeso J.A., Bezard E. and Vila M. Lewy Body extracts from Parkinson’s Disease Brains trigger and#945;and#8208;Synuclein Pathology and Neurodegeneration in Mice and Monkeys. Annals of Neurology (2013) [Epub ahead of print]
2. Perier C., Bender A., Garcíaand#8208;Arumí E., Meliŕ M.J., Bové J., Laub C., Klopstock T., Elstner M., Mounsey R.B., Teismann P., Prolla T., Andreu A.L. and Vila M. Accumulation of mitochondrial DNA deletions within dopaminergic neurons triggers neuroprotective mechanisms. Brain (2013) 136:2369and#8208;78
3. Ramonet D., Perier C., Recasens A., Dehay B., Bové J., Costa V., Scorrano L. and Vila M. Optic atrophy 1 mediates
mitochondria remodeling and dopaminergic neurodegeneration linked to complex I deficiency. Cell Death and Differentiation (2013) 20(1):77and#8208;85